Pharmaceutical Raw Phenytoin Sodium Phenytoin
Product Description

Product Details
Product Name |
Phenytoin |
Appearance |
Phenytoin Powder |
Assay |
>99% |
MW |
274.255 |
MF |
C15H11N2NaO2 |
Melting point |
290-299C |
Boiling point |
428.2C |
Phenytoin sodium is an antiepileptic and antiarrhythmic drug.
Phenytoin sodium can prevent abnormal electrical activity in brain lesions from spreading to surrounding normal brain tissue, playing an antiepileptic role. It has an inhibitory effect on ectopic rhythm points in the ventricles and atria, and can also accelerate conduction between the ventricles and atria, reducing myocardial autonomy, thus having an anti arrhythmic effect. Suitable for treating generalized tonic clonic seizures, simple and complex partial seizures, secondary generalized seizures, and status epilepticus in epilepsy. It can be used for conditions such as trigeminal neuralgia, paroxysmal choreographic hyperkinesia, paroxysmal control disorders, myotonia, and cardiac conduction disorders caused by excessive use of tricyclic antidepressants. Phenytoin sodium stabilizes the neuronal cell membrane by increasing the efflux of sodium ions through the cell membrane or decreasing the influx of sodium ions through the cell membrane, and limits the diffusion of episodic discharges.
Application&Function
The mechanism of action of Phenytoin includes:
Antiepileptic effect: Sodium phenytoin can prevent abnormal electrical activity caused by brain lesions from spreading to surrounding normal brain tissue, thereby exerting an antiepileptic effect. It is suitable for treating generalized tonic clonic seizures, simple and complex partial seizures, secondary generalized seizures, and status epilepticus in epilepsy. In addition, phenytoin sodium can also be used to treat conditions such as trigeminal neuralgia, episodic choreographic hyperkinesia, episodic control disorders, myotonia, and cardiac conduction disorders caused by excessive use of tricyclic antidepressants.
Antiarrhythmic effect: Phenytoin sodium has an inhibitory effect on ectopic rhythm points in the ventricles and atria, while accelerating atrioventricular conduction and reducing myocardial autonomy, thus having an antiarrhythmic effect. It can be used to treat arrhythmia caused by digitalis, restore normal sodium ion influx in Purkinje fibers, reduce abnormal ventricular autonomy, cell membrane reactivity, and refractory phase 2.
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